By Herman Friedman, Yoshimasa Yamamoto, Mauro Bendinelli
Chlamydia pneumoniae is a micro organism that's most typically identified for inflicting colds and pneumonia. besides the fact that, researchers have lately came across a hyperlink among C. pneumoniae and atherosclerosis, a clogging of the arteries that motives middle assault and stroke. additionally, ongoing study is exhibiting that yes lines might play a job in bronchial asthma, a number of sclerosis, Alzheimer's affliction, and arthritis.
This quantity, a part of the Infectious brokers and Pathogenesis sequence, is an entire portrait of C. pneumoniae and what's at the moment identified approximately it.
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Extra resources for Chlamydia pneumoniae : infection and disease
5. Hatch, T. , and Silverman, J. , 1982, Adenine nucleotide and lusin transport in Chlamydia psittaci, J. Bacteriol. 150:662–670. 6. Read, T. , Brunham, R. , Gill, S. , Heidelberg, J. , Hickey, E. , Salzberg, S. , and Fraser, C. , 2000, Genome sequences of Chlamydia trachomatis MoPn and Chlamydia pneumoniae AR-39, Nucleic Acid Res. 28:1399–1406. 7. , 2000, Comparison of whole genome sequences of Chlamydia pneumoniae J 138 from Japan and CWL 029 from USA, Nucleic Acid Res. 28:2311–2314. 8. , 1991, Morphological studies of the association of mitochondria with chlamydial inclusions and the fusion of chlamydial inclusions, J.
11, 12) The EBs of the C. pneumoniae TW-183 and AR (TWAR) strains have a wide periplasmic space limited by a wavy outer membrane, creating “pear-shaped” profiles in thin sections (Fig. 4a,b). In contrast, the EBs of the KKpn strains, which were isolated in Kawasaki Medical School Hospital and number from 1 to 18, were found to be round in shape with a narrow periplasmic space (Fig. (9, 13–15) This morphological difference between the TWAR and KKpn strains was also distinct when purified EBs of both strains were air-dried and then shadowcast (Fig.
Immunofluorescence microscopy of Hep2 cells infected for 48 h with C. pneumoniae cultivated on coverslips. After methanol fixation the sample was incubated with a monoclonal antibody recognizing the C. pneumoniae surface(5,13) and a polyclonal antibody raised against recombinant C. pneumoniae IncA. Secondary antibodies were FITC-conjugated goat anti-rabbit and rhodamine-conjugated goat anti-mouse antibodies. The first part shows IncA lining the inclusion membrane (FITC) and the second part the C.